Dietary supplement strengthening the body in prevention of Lyme disease, as well as supporting the direct fight against the disease. Opti Borelia® is a composition of highly concentrated plant extracts that not only boost the immunity, but also stimulate the body\'s cleansing of toxins produced by Borrelia bacteria. It also has a strong effect in other infections, not only bacterial.
The content of ingredients in a daily serving of product 1 capsule 2 capsules
Opti Borelis ® (proprietary formula) 100 mg 200 mg
Wild teasel root extract 10:1 (Dipsacus sylvestris ) 80 mg 160 mg
Herb of cistus extract 10:1 (Cistus Incanus L) 70 mg 140 mg
Bark of Vilcacora extract 10:1 (Uncaria tomentosa L) 60 mg 120 mg
Fallopia japonica extract 10:1 (Polygonum cuspidatum L.) 50 mg 100 mg
Andrographis paniculata extract 10:1 40 mg 80 mg
It's recommended to take 1-2 capsules a day with a water. 60 vegetarian capsules. A detailed description of the extracts can be found in our DiagnoHERB search engine - at www.naturday.pl/search or in the description below.

Borrelia burgdorferi.
The bacteria that is hiding.

Borrelia burgdorferi bacteria responsible for the development of the disease, opposite to many other bacteria, occurs in several forms: as spirochete, form L (without a cell wall) and cyst. This is the so-called polymorphism. Each form is sensitive to another type of antibiotic. It has also been shown that they have the ability to transform from one form into another. They only need a few hours to do it. The form that the bacteria takes depends on many factors, including the environmental conditions in which the bacterium is located. Bacteria with blood travel to the connective tissue (tendons, skin and blood vessels), muscles and nervous tissue. They are found numerous in the thalamus, dorsal roots of the spinal cord, dental pulp and eyeballs. The variety of bacterial forms makes it very difficult to eradicate, and various types of antibiotics should be administered during treatment.

Penicillins and cephalosporins work best for controlling spirochetes. The cysts are destroyed under the influence of metronidazole and tinidazole, and the L-form can be neutralised with tetracyclines or macrolides.

Spirochetes produce energy through the anaerobic breakdown of glucose, so during the treatment, additional oxygenation of the body is used. Despite the knowledge of bacteria-harmful antibiotics, the treatment is long-lasting mainly due to the ability of the bacteria to transform when the body contains antibiotics destroying the certain form. For this reason, during the treatment, a lot of antibiotics are used to minimise the risk of survival of any of the forms of bacteria despite treatment.
The other hang-up is the fact that antibiotics do not threaten a bacteria in the resting state. The bacteria can be destroyed only when they start to divide, creating new cell walls. The bacterium Borrelia distribution time is 12 to 24 hours, while for other bacteria a dozen minutes will do. The spirochaetes, cysts and L-forms can survive in dormancy for many years, waiting for the favourable environment and causing relapses of the disease, or during pregnancy, they can attack the child\'s system. Untreated Lyme disease is rarely the direct cause of death, but it clearly reduces the quality of life. Disease symptoms are related to a specific spirochaete species: e.g.: B. burgdorferi causes arthritic effects affecting the joints of the body; B. garinii is the cause of neurological symptoms; B. afzelii causes chronic skin symptoms. The cause of the disease is a bacterial toxin, which increases over time. The bacterium assumes microscopic cysts that are filled with toxin and bacterial spore-forming. The cysts have the ability to open up, releasing the bacterial endospores which later take the form of mature spirochaete. Even closed cysts release a toxin that irritates surrounding tissues, causing the intensity of symptoms.


In the first stage, the presence of specific IgM or IgG antibodies (depending on the clinical form) should be proven by immunoenzymatic method. In the second stage, patients with positive or doubtful results should be labelled with Western blot technique. Both methods complement each other.
A positive result of a serological examination without clinical symptoms typical for Lyme disease do not entitle to diagnose the disease or implement of appropriate treatment.
According to Public Health England (source: www.gov.uk), Laboratory reports of Lyme disease in England and Wales increase each year, In 2009 there were 863 reported cases, then 1040 in 2012 and 1579 in 2017. Lyme disease is more common in Poland, In 2009, 10,313 cases of Lyme disease were reported, and 8255 cases were reported a year earlier. Epidemiological reports in 2016 recorded the highest number: 21,220 officially reported cases, almost 56% more than registered in 2015 (13 625) and ten times more than at the beginning of this century.

Lyme borreliosis

Lyme disease, tick spiral disease, is a multisystemic infectious disease caused by bacteria belonging to the spirochetes: Borrelia burgdorferi, Borrelia garinii, Borrelia afzelii, Borrelia japonica, transmitted to humans and some animals by ticks. Lyme disease is the most common tick-borne disease. Human infection occurs through saliva or tick vomit. Numerous animal species, mainly rodents, are the reserve of the spirochete.

Early symptoms of the disease include characteristic erythema characterised by peripheral dissemination and central regression. Erythema migrans is the most common symptom of early Lyme disease. Other early lesions include rare lymphocytic infiltration of the skin and flu-like symptoms including fever, headache, weakness. Early symptoms subside within 3 months and in some patients an early dissemination develops, which may occur in the form of arthritis or neuroborreliosis or (less often) myocarditis.

After many years of the first infection, some patients develop atrophic dermatitis, chronic arthritis and neurological complications. Lyme disease is diagnosed on the basis of positive serological tests and the occurrence of typical clinical symptoms. The infection is effectively treated with antibiotics from the group of penicillins, cephalosporins and tetracyclines.
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1. Erythema migrans (EM) - is the most common symptom of Lyme disease. Within 1-3 weeks after the tick bite, a single erythematous spot or papule appears on the skin, exactly in place of the bite, which quickly expands with the red ring around the central spot.
2. Borrelial lymphocytoma (BL) lymphocytic lymphoma [lymphadenosis benigna cutis] Reveals in less than 1% of patients, usually a few weeks after being bitten by a tick, as a single, blue-red, non-painful lump. The BL location is the ear auricle, scrotum and nipple.
2a. chronic atrophic dermatitis (acrodermatitis chronica atrophicans) - reddish-bluish, usually asymmetrical skin changes in further parts of the limbs that appear several years after infection (late stage); initially have the character of inflammatory oedema, but later the features of disappearance dominate - a thin and hairless skin with a purple discolouration.
3. Arthritis (LA - Lyme arthritis).
Migrating pain of bones, joints, muscles or tendon can occur at the early stage of multiple infection, within a few weeks of contracting. Recurrent arthritis (usually asymmetric), where the pain is accompanied by swelling and increased warming, usually appears within two years after infection. In case of a large exudate in the joint cavity, the pain intensify, especially when moving the limbs. Frequently the changes range the knee joints, and less frequently a shoulder, shoulder joints, elbow joints, wrist joints, hip joints and ankle joints. Much less frequentlyt hey concern temporomandibular joints and minor joints of the hands and feet.
Untreated Borella burgdoferi infection can last for months or even years leading to permanent, irreversible damage to the affected organs.
4. Neuroborreliosis
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1) lymphocytic meningitis - usually with a mild course,
the only symptom can be a headache
(early disseminated stage)
2) cranial nerves inflammation - paralysis or paresis (most often) of the facial nerve, (Bell's paralysis, can be bilateral, Lyme disease is the most common cause. Llymphocytic meningitis along with peripheral neuropathy are the Bannwarth syndrome (other names: Lymphocytic meningoradiculitis, meningo-polyradiculoneuritis), early disseminated stage.
3) radiculitis (radiculoneuritis) and peripheral neuropathy - is manifested by strong neuralgia increasing at night and peripheral neuropathy (paresthesia, muscle weakness) usually within one or several dermatomes; in late neuroborreliosis is the dominant symptom
5. Myocarditis (LC - Lyme carditis). The most common are conduction disorders in the form of variable-degree anterograde blocks.

6. Post-Lyme disease syndrome. This refers to a series of symptoms persisting in patients with Lyme disease after treatment. The main symptoms are fatigue and joint and muscle pain. In some cases, it may take up to 6 months. The exact reason for this syndrome has not been yet diagnosed. Most experts believe that it is the result of tissue damage and the immune system arisen during the infection. Similar symptoms occur as a result of other infections, such as: Campylobacter (Guillain-Barré syndrome), Chlamydia (Reiter's syndrome) or Streptococcus (rheumatic fever). Studies have not shown that the condition of patients receiving prolonged antibiotic therapy improves faster than those who receive a placebo. In addition, long-term antibiotic therapy carries the risk of serious complications.
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The prognosis in the course of properly treated Lyme disease is usually good. 70-98% of patients with erythema migrans, return to full health within 12 months of antibiotic therapy. In the case of neuroborreliosis in 70-85% of patients, the symptoms disappear within 6-12 months after the treatment inclusion. When joint symptoms persist for more than 3 months after antibiotic therapy, antibiotic-resistant Lyme disease should be diagnosed. It affects about 10% of patients. Symptoms may persist for several years. Non-steroidal anti-inflammatory drugs, intraarticular corticosteroid injections or synovectomy [4,64,102-104] are helpful in the treatment. About 5% of patients with Lyme disease suffer from post-Lyme disease syndrome (PLDS).

1. Borelioza z Lyme. prof. dr hab. Robert Flisiak, prof. dr hab. Jacek Szechiński. Interna Szczeklika – Podręcznik chorób wewnętrznych 2014, rozdz. 11 Wybrane zagadnienia chorób zakaźnych.

https://www.mp.pl/okulistyka/okulistyka-dla-nieokulistow/artykuly/122304,borelioza-z-lyme (access 09.03.2018)

2. Diagnostyka i leczenie boreliozy z Lyme. Robert Flisiak, Klinika Obserwacyjno-Zakaźna AM w Białymstoku Sławomir Pancewicz,Klinika Chorób Zakaźnych i Neuroinfekcji AM w Białymstoku. https://infekcje.mp.pl/wytyczne/51646,diagnostyka-i-leczenie-boreliozy-z-lyme (access 09.03.2018)

3. Przegląd Lekarski 2015 / 72 / 11 REVIEW BOOKS - Borelioza – aktualny stan wiedzy. Justyna Błaut-Jurkowska Marcin Jurkowski http://www.wple.net/plek/numery_2015/numer-11-2015/656-661.pdf (access 09.03.2018)

4. Borelioza - niebezpieczna choroba z Lyme. Zuzanna Koperwas Ogólna charakterystyka i diagnostyka laboratoryjna zakażenia. http://laboratoria.net/artykul/15100.html (access 09.03.2018)


  • 30% ticks are infected with Borrelia bacteria.
  • improperly treated Lyme disease becomes the cause of disability or even death.
  • The synergy of action of antibiotic therapy and extracts of natural herbs and medicinal plants is more effective in the treatment of LYME DISEASE and the proactive use of herbs, or actually, their extracts improve the immunity and can prevent the disease.


The composition of natural plant extracts - herbs, that support the fight against the Lyme disease. It contains active ingredients with powerful anti-bacterial properties (against Spirochaetes), that also support and stimulate the immune system.
Lyme disease is not easy to fight, its therapy requires the use of significant doses of antibiotics, prescribed by the doctor. In addition to basic antibiotic therapy, it is helpful to support the treatment process and your body condition with a proper diet and natural supplementation, such as the Opti Borelia®. It is worth remembering that the unique OptiBorelia® composition was created thanks to the work of experienced pharmacognosy experts and contains the highest quality, high-concentration of natural plant and herb extracts. Thanks to this, we achieve the expected effect of supplementation much faster. The use of the maximum quality raw material means that the price of the composition can sometimes be higher than other common herbal products, but the vital importance in the highly purified and concentrated plant extracts used in OptiBorelia® and other NaturDay® products is receiving much better effects, which ultimately turns out to be one of the fundamental reasons for our customers' satisfaction.


Opti Borelis®

The proprietary formula contained in the product helps to stimulate the immune system to fight the Lyme disease and has a strong antioxidant effect.

Wild teasel (Dipsacus fullonum)

(Dipsacus sylvestris) Wild teasel root extract (Dipsacus sylvestris) (DER 10:1; extractant: water and ethanol). Wild teasel root extract is useful in strong and long-term infections, used in Lyme disease therapy. This extract effectively destroys bacteria, protozoa, and some fungi, its effectiveness is compared to the treatment of amoxicillin, which is using to eradicate Lyme disease. Properties: Bacteriostatic function, protective effects on liver parenchyma (hepatoprotective), anti-inflammatory, analgesic, anti-rheumatic and immunostimulant. The raw materials are rich in chlorogenic acid, quinic acid and caffeic acid, also contain essential iridoids, triterpenoid (ursolic acid), saponins and alkaloids. The effects on Lyme disease: bacteriostatic, toning for the liver and kidneys, strengthening tendons and bones, anti-inflammatory in conditions related to the back pain and limb pain (especially knees), antalgic in painful swellings and improving blood circulation. Uses: We can use the wild teasel root in infections and in convalescence processes after chronic diseases connected with weight loss.

Quotation: https://www.ncbi.nlm.nih.gov/pubmed/?term=Dipsacus+sysvestris,https://www.ncbi.nlm.nih.gov/pubmed/21901989 http://rozanski.li/345/borelioza-borreliosis-fitoterapia-cz-iii-i-troche-narzekania/


(Cistus Incanus L). Herb of cistus extract (Cistus Incanus L). (DER 10:1); extractant: water and ethanol, Herb of cistus extract used in the fight against infections, strengthens the immune system. Contain lots of polyphenols which are strong antioxidants. Cistus also helps to detox the system, for example by removing heavy metals from the body. Properties: Herb of cistus is marked by a high content of polyphenols (supposedly even greater than red wine) - strong antioxidants. These substances have antioxidant properties, i.e. neutralise free radicals, which can contribute to the formation of many diseases, including cancer. They also inhibit the formation and development of inflammation in the body, strengthen and add energy, protect blood vessels and inhibit the oxidation of "bad" LDL cholesterol, preventing the formation of atherosclerotic plaques. They also support the functioning of the immune system and fight against contagium and fungi, they also have antiallergic properties, because they inhibit the release of histamine. Uses: The herb and leaves of cistus were used from centuries in skin diseases, rheumatism and nephritis. They were also valued for their anti-ulcer, anti-diarrheal and healing properties. These raw materials are also gaining popularity in European medicine. Infusions of herb or leaf are used in the prevention of various diseases. Currently, herb and purge leaves are recommended as antimicrobial, antiviral, antifungal, as well as strengthening the immune system. The antioxidant activity resulting from the composition of raw materials is considered the most important profile of biological activity. They are recommended as dietary supplements in the prevention of chronic diseases such as hypertension, diabetes or Alzheimer's disease. The European Food Safety Authority (EFSA) recommends these raw materials as a rich, natural source of antioxidant substances. Research is continuing, which confirms well-known traditional applications, as well as show new profiles of the biological activity of extracts from these raw materials and secondary metabolites isolated from raw materials. Spasmolytic properties (in the treatment of diarrhoea and diseases of the digestive system) and hypotensive properties were supported by scientific research. It has been proven that aqueous extracts of C. Incanus leaves in vitro, relax the isolated smooth muscle of the intestines and aorta.

Quotation Dr Henryk Różański. Studies: Reflections on purges, (original: Rozmyślania nad czystkiem). 28.05.2014

Cat’s claw. Vilcacora.

(Uncaria tomentosa L.) Bark of Vilcacora/Cat\'s claw extract (Uncaria tomentosa L) (DER 10:1; extractant: water and ethanol). Cat’s claw extract contains alkaloids that stimulate white blood cells, which destroys bacteria that attack the system, it has anti-inflammatory, antioxidant and antiviral properties. It’s recommended for people with weak immunity, for example after radiotherapy or chemotherapy. Properties: Immunostimulating effect; stimulating phagocytosis (antigen devouring), increasing the number of T, NK and cytotoxic lymphocytes; stimulating the GALT and MALT system; calming \'antiviral, especially against HIV; adaptogenic (improves adaptability of the body), anti-inflammatory, anti-rheumatic, diastolic blood vessels and respiratory tract, anti-asthmatic; antioxidant; protective against liver parenchyma and antimutagenic. Active ingredients found: Tetra- and pentacyclic oxindole alkaloids (about 2%), triterpene saponins, phenol acids (quinic acid), tannins. There are two plant chemotypes: with predominant pentacyclic alkaloids and with predominant tetracyclic alkaloids. The chemotype with pentacyclic alkaloids is more valuable, especially in the treatment of AIDS and cancer, moreover viral liver inflammation and rheumatic diseases. There are various chemotypes on sale and usually, the patient is unaware of what raw material he buys, hence the variance of effectiveness. There are raw materials and ready-made preparations standardised for the content of pentacyclic alkaloids (pteropodine, mitrafilin) and tetracyclic (ergophylline). Uses: Viral diseases; immunodeficiency, cancer (adjuvant); rheumatic diseases; disorders of the organism\'s adaptation to the environment.

Quotation: https://www.ncbi.nlm.nih.gov/pubmed/?term=Uncaria+tomentosa https://www.ncbi.nlm.nih.gov/pubmed/29442306http://rozanski.li/2021/uncaria-tomentosa-willdenow-de-candolle-czepota-puszysta-w-fitoterapii/

Fallopia japonica.

(Polygonum cuspidatum L.) Japanese knotweed (Polygonum cuspidatum L.). (DER 10: 1; extractant: water and ethanol) The extract of Japanese knotweed acts antibacterially against the Lyme spirochete and additionally stimulates the immune system to fight bacteria and viruses. Pollen is a source of resveratrol, which stimulates microcirculation, regulates metabolism, selenium, copper, manganese, iron and zinc. In the rhizomes there are flavonoids: quercetin (quercetin), luteolin (luteolin), apigenin (apigenin), kemferol (kaempherol = kaempferol); anthraquinone and naphtoquinone compounds (glycofrangulin (glucofrangulin), 2-methoxy-6-acetyl-methyl-juglone, chrysopanol = 1.8-dihydroxy-3-methylanthraquinone, anthraquinone - emodine (1,3,8-trihydroxy-6-methylanthraquinone) ; parietine = parietin, i.e. reochiridine = rheochrysidin (1,8-dihydroxy-3-methoxy-6-methylanthraquinone), questin = polytin (emodin-8-methyl ether), fallacinol = 1,8-dihydroxy-3- (hydroxymethyl) -6-methoxy-9,10-anthracenedione; stilbenes: resveratrol - resveratrol (5-trihydroxystilbene); polidatin = polydatin (resveratrol 3-O-beta-D-glucoside); phenolic acids, phenols: protocatechin acid (3,4- dihydroxybenzoic acid), gallic acid (3,4,5-trihydroxybenzoic acid), coumaric acid (cis-o-hydroxycinnamic acid), methyl coumarin, catechinic acid and protocatechin; phytosterols: glycosidically bound beta-sitosterol, stigmast-5-ene-3β 7α-diol, daucosterol, cycitoles (mannitol). Products from knotweed are a source of bioelements: selenium, copper, molybdenum, mang an, iron, zinc. Resveratrol, polydatine and naphtoquinones inhibit the growth of fungi and pathogenic bacteria. They belong to natural chemopreventive factors in relation to cancer. Hydroxystilbene, like polyphenols (including bioflavonoids) are scavengers of free radicals, antioxidants, thus stabilizing the structure of cell membranes. In vitro, they are inhibitors of carcinogenesis (inhibit the development of tumors). They activate quinone reductase and inhibit the activity of peroxidase prostaglandin cyclooxygenase I. They induce apoptosis (programmed cell death). They inhibit the aggregation of blood cells, preventing their adhesion to protein fibers and calcium absorption in thrombocyte conglomerates. Thanks to this, they reduce the risk of blood clots. Tetanus bioflavonoids and hydroxystylbenzenes are also ACAT-acetyl-CoA-cholesterol acetyltransferase inhibitors, an enzyme responsible for esterification and thereby cholesterol absorption. ACAT inhibitors reduce the concentration of cholesterol esters in atherosclerotic plaques and reduce the size of atherosclerotic plaques. They have antiatherosclerotic properties. Resveratrol and polidatine enhance the synthesis of nitric oxide, which is an endogenous mediator that dilates blood vessels and improves blood flow. They are also tyrosine kinase (tyrosine kinase) inhibitors that catalyze tyrosine phosphorylation. In this mechanism of action of resveratrol, its antimutagenic properties are seen. By inhibiting cyclooxygenase and lipoxygenase, it reduces inflammation and exerts analgesic effects. Phytosterols, polyphenols and hydroxystilbena prevent fatty and fibrosis of the kidneys, liver and heart (lipotropic effect). They decrease elevated levels of aspartic and alanine aminotransferase. Polydatin knotweed reduces the risk of damage to myocardium cells in the event of a temporary lack of glucose and oxygen. It also prevents side effects caused by the administration of anti-hypertensive drugs (against hypertension, e.g. clonidine) and neuroleptics (eg chlorpromazine). Application: coronary disease, diabetes, hypertension, inflammation of veins, atherosclerosis, peripheral circulation disorders, clots, cancer phytotherapy, autoimmune diseases, constipation, intestinal atony, poisoning, detoxification treatments, obesity, memory disorders, viral hepatitis, cirrhosis, disease Alzheimer\'s syndrome, cold hands and feet syndrome (eg young girls), menopause, prostatic hypertrophy, inflammation and urinary and sexual system infections (in mixes), stress, depression, convalescence after suffering infectious diseases, chronic skin diseases on the background of disorders metabolism, prophylaxis and treatment of candidiasis (Candida), parasitic diseases, inflammation of the cornea, iris and choroid, cataracts, bronchial asthma, inflammation of the breast.

Quotation: http://rozanski.li/4352/rdestowiec-reynoutria-fallopia-w-praktycznej-fitoterapii/ https://vsearch.nlm.nih.gov/vivisimo/cgi-bin/query-meta?query=polygonum+cuspidatum&v%3Aproject=nlm-main-website https://vsearch.nlm.nih.gov/vivisimo/cgi-bin/query-meta?query=polygonum+cuspidatum&v%3Aproject=nlm-main-website https://www.ncbi.nlm.nih.gov/pubmed/?term=polygonum+cuspidatum

Andrographis paniculata

(Andrographis paniculata) Andrographis paniculata leaf extract (Andrographis paniculata (Burm.f.) Wall. ex Nees.) (DER 10:1; extractant: water and ethanol) - an antibacterial and antiparasitic herb that shows the strongest effect in lowering the inflammatory cytokine 1L-1b against Lyme disease. It strengthens immunity, accelerates digestion and cleanses the body of toxins. Properties: antibacterial, antifungal and antiviral, against cold, sinus infection, general inflammation, pain relief and detoxification. Bioactive substance: diterpenoid (andrographolide); andrografis, mistress. Usage: It shows activity against viruses, bacteria and parasites and it has antioxidant properties; protects the body\'s cells, including neurons (neuroprotective effect). Diseases of joints and bones, skin ulcers and mucous membrane ulcers, cough, diarrhoea, dysentery, boils and painful urination. Digestive disorders, lack of appetite, diseases of the pancreas, liver and spleen disorders; inflammation of the sinuses, allergic diseases; infections of the respiratory and genitourinary systems, autoimmune diseases, hyperandrogenism. ; Andrographis paniculata is a plant classified as an immunomodulator.

Quotation: https://cmjournal.biomedcentral.com/articles/10.1186/17498546517, https://www.ncbi.nlm.nih.gov/pubmed/19752167 https://www.ncbi.nlm.nih.gov/pubmed/29408515, https://en.wikipedia.org/wiki/Andrographis_paniculata https://www.ncbi.nlm.nih.gov/pubmed/?term=Andrographis+paniculata http://rozanski.li/3441/andrographis-green-chirettta-indian-chiretta-w-praktycznej-fitoterapii/

Garlic (Allium sativum L.)

(Allium sativum L.) Extract of garlic, obtained and combined in a particular way with extract of cistus (rockrose) makes the Opti Borelis®. This proprietary formula - helps to stimulate the immune system to fight the Lyme disease and has a strong antioxidant effect. Properties: The main active substance is alliin, which is broken down by the enzyme alliinase mediated by the product unstable to allicin (in processed materials it\'s often the main ingredient). In garlic, an alliinase enzyme acts on the chemical alliin converting it into allicin. Further metabolites are inhibitors of platelet aggregation (increase bleeding time, anticoagulant). Garlic increases the secretion of gastric juice, at the same time protects against heartburn, it has a cholagogic and antispasmodic properties, and it has a beneficial effect on the respiratory tract, regulates the bacterial flora and conduces to the better blood supply to the coronary vessels. It acts as a mild antibiotic with its antibacterial, antifungal and anthelmintic properties. The most sensitive to a garlic oil are strains of anaerobic bacteria of the genus Bacteroides, Prevotella, Fusobacterium and Actinomyces. It also works on streptococcus Streptococcus, staphylococcus staphylococci, mycobacteria causing tuberculosis and colon bacteria. The least sensitivity to garlic oil is demonstrated by Veillonella and Porphyromonas strains. Essential oil found in garlic works effectively in low concentrations on anaerobic bacteria involved in infections of the mouth and upper respiratory tract. It combats the urinary tract infections. Antifungal activity is attributed to the ability to inhibit the synthesis of lipids in the mycelium. Garlic extract is recommended as an additional component to the toothpaste or mouthwash. Garlic works prophylactically and therapeutically also in lead poisoning. Used externally for the enema in the treatment of pinworm infection and irrigation in case of Trichomonas vaginalis infection. Fresh juice also inhibits the development of influenza viruses. It has a diaphoretic effect, reduces fever and blood pressure. Reduces inflammation. It\'s an antioxidant that protects the liver. In addition, it lowers the level of PG2 prostaglandin, reducing pain during menstruation. Also described as an anti-atherosclerotic agent that reduces blood cholesterol. This last property has been proven in animal tests, as well as described from human research, but it has not been confirmed by comprehensive studies conducted in the United States. Extensive research and review of scientific sources do not confirm or deny the desirability of using garlic in the clinical treatment of hypertension, and their results are contradictory. Czosnkowi przypisuje się także działanie antysklerotyczne. Udokumentowane klinicznie działanie - obniżające lipidy, hamujące agregację trombocytów, fibrynolityczne, antybakteryjne, lekko obniżające ciśnienie krwi. Zastosowanie: Infekcja wirusowa, Infekcja bakteryjna, Zakażenie pasożytami, Miażdżyca, Nadciśnienie., Zapobieganie - Zakrzepica (zatory, udar niedokrwienny mózgu, zawał serca). Garlic is also credited with anti-sclerosis properties. There is a clinically documented action of lipid-lowering, thrombocyte aggregation-inhibiting, fibrinolytic, anti-bacterial, slightly lowering blood pressure. Uses: Viral infection, bacterial infection, parasite infection, atherosclerosis, hypertension, prevention - thrombosis (congestion, ischemic stroke, myocardial infarction).
References: dr n. farm. Jadwiga Nartowska. Panacea Nr 1 (18), styczeń - marzec 2007 strony: 8-11. Czosnek, a choroby układu krążenia. lek. med., mgr farm. Jarosław Struczyński. Panacea Nr 1, październik 2002 strony: 28-30. Czosnek - Antybiotyk starożytności. Encyklopedia internetowa Wikipedia. Czosnek pospolity. dostęp 07.03.2108., Jerzy Lutomski. Borgis - Postępy Fitoterapii 1/2001, s. 7-14. Fascynacja czosnkiem – wczoraj i dziś.Krzysztof Marciniec1, Barbara Włodarczyk-Marciniec2 Borgis - Postępy Fitoterapii 2/2008, s. 90-95. Przeciwnowotworowe własności czosnku. Anna Kędzia. Borgis - Postępy Fitoterapii 1/2010, s. 46-52. Przeciwdrobnoustrojowe działanie czosnku ( Allium sativum L.) Polecana literatura. Burdzenia O. Impresje na temat czosnku. "Wiadomości Zielarskie", nr 17/2000; Encyklopedia zielarstwa i ziołolecznictwa pod red. H. Strzeleckiej i J. Kowalskiego. W-wa 2000; Hasik J., Lutomski J. Ziołolecznictwo w chorobach wewnętrznych. Racjonalny poradnik dla lekarzy i farmaceutów. W-wa 2000; Kawałko M. J. Historie ziołowe. Lublin 1986; Kędzia A. Działanie olejku czosnkowego na bakterie beztlenowe wyodrębnione z jamy ustnej i górnych dróg oddechowych. "Postępy Fitoterapii", nr 1/2000; Klimuszko A. Cz. Wróćmy do ziół. W-wa 1989 (wyd. III); Khan Z. H., Katiyar R. Potent anti-fungal activity of garlic (Allium sativum) against experimental murine disseminated cryptococcosis. "Pharmaceutical Biology" 2000, 308, 87-100; Kohlmünzer S. Farmakognozja. W-wa 2000; Lutomski J. Czosnek znany i nieznany. W-wa 1989; Lutomski J. Fascynacja czosnkiem - wczoraj i dziś. „Postępy Fitoterapii", nr 1/2001; Medycyna naturalna pod red. K. Janickiego i W. Rewerskiego. W-wa 2001; Niedworok J. Lecznicze właściwości czosnku odkrywane na nowo. "Wiadomości Zielarskie", nr 11/2000; Siegel G. i inni. Pleiotropowe działanie czosnku. "Herba Polonica", nr 3/1999. Adler R., Lookinland S., Berry J.A. A systematic review of the effectiveness of garlic as antihyperlipidemic agent. J. Am. Acad. Nurse Pract., 2003,15,120-129; Banerje Sajany K., Malik Subir K.: Effect of garlic on cardiovascular disorders: a review. Nutrition Journal, 2002,1(1); 4-32; Beaglehole R. Cardioprotection and garlic. Lancet,1997, 349:131; Birdia A., Verma S.K., Srivastava K.C.: Effect of garlic (Allium sativum) on blood lipids, blood sugar, fibrinogen and fibrinolytic activity in patients with coronary artery disease. Prostaglandins, Leukotrienes and Essential Fatty Acids, 1998,58 (4), 257-263; Bruneton J. Pharmacognosy, Phytochemistry Medicinal Plants. Lavoisier, Paris, London, New York 1999; Efendy J.L., Simmons D.L., Campbell G.R., Campbell J. The effect of the garlic extract „Kyolic”, on the development of experimental atherosclerosis. Atherosclerosis, 1997, 132, 37-42; Kiesewetter H., Jung F., Jung E. M., Mrowietz C., Kościelny J. Effect of garlic on platelet aggregation in patients with increased risk of juvenile ischaemic attack. Eur.J. Clin. Pharmacol. 1993, 45, 333-336; Kiesewetter H., Jung F., Pindur G.Jung E. M., Mrowietz C., Wenzel E. Effect of garlic on thrombocyte aggregation, microcirculation, and other risk factors. International J. of Clinical Pharmacology, Therapy and Toxicology, 1991, 29 (4), 151-155; Mader F.H. Treatment of hyperlipidaemia with garlic-powder tablets. Arzneim.Forsch./Dru Res., 1990, 40 (11), 1111-1116; Matławska I. Farmakognozja. Ak. Med., Poznań 2005;
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Kiesewetter H., Jung F., Pindur G.Jung E. M., Mrowietz C., Wenzel E. Effect of garlic on thrombocyte aggregation, microcirculation, and other risk factors. International J. of Clinical Pharmacology, Therapy and Toxicology, 1991, 29 (4), 151-155; Mader F.H. Treatment of hyperlipidaemia with garlic-powder tablets. Arzneim.Forsch./Dru Res., 1990, 40 (11), 1111-1116; Matławska I. Farmakognozja. Ak. Med., Poznań 2005; Morris J., Burke V., Mori T.A., Vandongen R., Beilin L.J. Effects of garlic extract on platelet aggregation: a randomized placebo-controled double-blind study. Clinical and Experimental Pharmacology and Physiology 1995, 22, 414-417; Morihara N., Ushijima M., Kashimoto N., Sumioka I. Aged garlic extract ameliorates physical fatigue. Biol. Pharm. Bull. 2006, 29(5), 962-966; Patumraj S., Tewit S., Amatyakul S., Jariyapongsul A. Maneesri S., Shepo D. Comparative effects of garlic and aspirin on diabetic cardiovascular complications. Drug Delivery 2000,7, 91-96; Rahman K. Garlic and aiging: new insights into an old remedy. Ageing Research Reviews 2003, 2, 39-56;